ER stress implication in the fibrotic process has been largely documented in many organs. We propose to specially document the implication of UPR actors on the induction of lung pro-fibrotic signaling pathways at the tissular and cellular level.
ER stress implication in the fibrotic process has been largely documented in many organs. We propose to specially document the implication of UPR actors on the induction of lung pro-fibrotic signaling pathways at the tissular and cellular level.
